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HOME > J. Microbiol > Volume 53(11); 2015 > Article
Research Support, N.I.H., Extramural
The Mycobacterium tuberculosis relBE toxin:antitoxin genes are stress-responsive modules that regulate growth through translation inhibition
Shaleen B. Korch 1,2, Vandana Malhotra 1, Heidi Contreras 1, Josephine E. Clark-Curtiss 1,3
Journal of Microbiology 2015;53(11):783-795.
DOI: https://doi.org/10.1007/s12275-015-5333-8
Published online: October 28, 2015
1Center for Infectious Diseases and Vaccinology, Biodesign Institute, Arizona State University, Tempe, Arizona 85287, USA, 2Department of Pharmacology, Midwestern University, Glendale, Arizona 85308, USA, 3School of Life Sciences, Arizona State University, Tempe, Arizona 85287, USA1Center for Infectious Diseases and Vaccinology, Biodesign Institute, Arizona State University, Tempe, Arizona 85287, USA, 2Department of Pharmacology, Midwestern University, Glendale, Arizona 85308, USA, 3School of Life Sciences, Arizona State University, Tempe, Arizona 85287, USA
Corresponding author:  Shaleen B. Korch , Tel: +1-623-572-3708, 
Received: 2 July 2015   • Revised: 30 September 2015   • Accepted: 5 October 2015
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    The Mycobacterium tuberculosis relBE toxin:antitoxin genes are stress-responsive modules that regulate growth through translation inhibition
    J. Microbiol. 2015;53(11):783-795.   Published online October 28, 2015
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